Bodensteiner Medical Research
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Welcome to Thrombo.Info
by Dr Bode

The Happy Heart

Tick-tock, tick-tock
The happy heart beats like a clock.

 

Do you ever lie awake at night and wonder what makes the healthy heart flip and flop like a fish out of water? Suddenly the heart flutters like a humming bird and then slows down with powerful pounding palpitations that cause hot flashes and night sweats.

Doctors observe fast fluttering pulses followed by slow strong palpitations and call this condition the "sick sinus syndrome". Its cause is idiopathic, which means unknown.

Bodensteiner Medical Research (BMR) studies holter electrocardiogram (ECG) information and simultaneous pulse oximeter data on patients that suffer the sick sinus syndrome, which is also known as the "tachy-brady" or "brady-tachy" syndrome. Patients suffer brain fog, confusion, light-headed dizzy spells, and fainting episodes.

BMR applied sequential venous compression devices to sore legs of patients with phlebitis, and recorded the "tachy brady" syndrome using the holter ECG monitor.

BMR theorizes that compression of sore muscles full of deep venous thrombosis (DVT) squeezes bloody clots into venous circulation. As clots (venous thromboembolism / VTE) migrate into the right heart pump they alter blood flow at the valves and cause the sick sinus syndrome!

Thrombodextracardia describes blood clots inside the heart valves, which cause premature heartbeats that make up the sick sinus syndrome.

First, clots cause fast atrial contractions as they go into the right ventricle through the tricuspid valve. Next, they cause slow skipped heartbeats called "premature ventricular contractions" (PVC) as they pass through the pulmonary valve and move into the pulmonary artery.

Thrombodextracardia is a new theory that explains the sick sinus syndrome: fast fluttering premature atrial beats plus slow flip-flop PVC's equals the idiopathic "tachy-brady" sick sinus syndrome.

Hemodynamic Electrocardiac Theory: A New Interpretation of the Electrocardiogram

  • The heart is composed of two pumps intimately connected like Siamese twins.
  • The heart has four valves that connect six pumping parts.
  • The six pumping parts include four chambers and two great arteries.
  • These connected pairs of pumps have sequential synchronized rhythmic contractions.
  • First: two atria contract together and fill up the ventricles.
  • Next: two ventricles contract together and fill up two great arteries.
  • Last: two great arteries contract and pump blood away from the heart.
  • In 1903, Dr Einthoven discovered that the heartbeat generates electricity.
  • The heartbeat electrocardiogram (ECG) has three waves: P, QRS, and T.
  • Action potential depolarization / repolarization explain ECG physiology.
  • Electric action potentials move at the speed of light, which blurs the evidence.
  • Bodensteiner Research reinterprets the ECG in terms of blood flow.
  • Blood flow generates friction electrical potentials!
  • Normal atrial contractions generate friction blood flow P waves.
  • Normal ventricular contractions generate blood flow QRS waves.
  • Aortic and pulmonary artery contractions generate blood flow T waves.
  • Hemodynamic friction electric potentials explain ECG physiology.
  • Current depolarization / repolarization ECG theories need revision.

Carboxyhemoglobin Pulse Oximetry Data leads to theory of venous stasis

New pulse oximeters measure the carboxyhemoglobin level. Venous acidosis caused by abnormal hypoxic metabolism changes the carboxyhemoglobin level, and there seems to be quantitative and qualitative relationships between SpCO levels (carboxyhemoglobin) and phlebitis in patients who suffer DVT's with sore weak warm red swollen muscles. Carboxyhemoglobin appears to be a circulating biomarker that evaluates slow venous blood flow, which causes hypoxic venous acidosis. Venous acidosis leads to blood clots, phlebitis, DVT's and peripheral neuropathy. Carboxyhemoglobin locates venous vascular stasis with hypoxic acidosis that seems to be the main basis of hemothrombosis. Carboxyhemoglobin (SpCO) is a biomarker of peripheral neuropathy and locates the origin of clots.

Thrombo Questions & Answers:

  • What makes the heart skip a beat?
  • How does a blood clot cause the heart to skip a beat? 
  • What causes blood to form clots? 
  • Why does abnormal hypoxic metabolism make lactic acid?
  • How does lactic acid activate the blood clotting mechanism?
  • What makes blood clots migrate (embolize) into heart valves? 
  • What happens to the heart rhythm as clots pass through different heart valves?
  • Do blood clots cause fluttering or flip flop palpitations?
  • Why do arrhythmias cause low blood pressure with lightheaded dizzy spells?
  • Does partly clotted blood called detritus interfere with breathing?
  • How does bloody debris (detritus) cause exercise-induced asthma?
  • How do blood clots cause coughing, nausea, gagging and sneezing?
  • Why do blood clots accumulate in the pulmonary artery next to the esophagus?
  • Do blood clots or detritus cause panic attacks or internal suffocation?
  • Do blood clots cause pulseless fainting?
  • How do blood clots cause epileptic seizures?
  • How do blood clots cause sudden thrombocardiac arrest?
  • How does CPR reanimate someone with cardiac arrest without defibrillation?

Thrombo Associated Diseases:

  • Cancer → lactic acid → blood clots (Warburg effect)
  • Carbon monoxide poisoning: night time hypoxemia → carboxyhemoglobinemia
  • Chronic Fatigue Syndrome
  • Congestive heart failure: right heart failure, valve malfunction, low ejection fraction
  • Exercise-induced asthma: pulmonary embolism (PE) of detritus
  • Infection: anaerobic germ metabolism muscle: glucose → lactic acid → blood clots
  • Injuries: foot, leg, knee, or hip injury → blood clots
  • Gastro esophageal reflux disease (GERD): emboli in pulmonary artery compress  the esophagus between heart and vertebra, stimulates burping during sleep → GERD 
  • Headaches: pulmonary detritus ↓ water exhalation → brain edema, headache
  • Insomnia: PE at tricuspid valve → thrombotachycardia → sleep arrousal 
  • Macular degeneration: micro emboli
  • Malignant hyperthermia: PE → atelectasis, fever, VTE arrhythmias
  • Migraine headaches
  • Narcolepsy: PE detritus → ↓ exhalation of CO2 → hypercapnea → CO2 narcosis
  • Panic attacks: PE of detritus, desaturation, dyspnea
  • Peripheral neuropathy: venous vascular acidosis → neuropathy, restless leg
  • Pleurisy: PE into alveoli causes sharp ischemic pain
  • Pre-menstrual syndrome: PE of detritus, hot flashes, headache (edema), mild fever
  • Seizures: VTE at pulmonary valve, sudden hypoxia, convulsions perform CPR
  • Sick Sinus Syndrome: VTE at tricuspid and pulmonary valves
  • Sleep apnea: PE detritus → internal hypercapnea with central CNS depression
  • Syncope: VTE at pulmonary valve, skipped heartbeats
  • Tinnitus: emboli of detritus into sensitive area of inner ear

Thrombo Symptoms

  • Anxiety, panic attacks
  • Brain Fog
  • Choking
  • Coughing
  • Confusion
  • Depression
  • Dizzy spells
  • Dyspnea (short of breath)
  • Exercise-induced asthma
  • Fainting
  • Fatigue
  • Fear of sudden death
  • Fever (fever seizures, night sweats, hot flashes)
  • Fluttering fast palpitations
  • Flip-flop pounding strong palpitations
  • Gagging
  • Headaches
  • Hot flashes
  • Insomnia with fast racing heartbeats
  • Irregular pulse
  • Irritability
  • Phlebitis
  • Premature beats
  • Light-headed sensations
  • Muscular dystrophy
  • Nausea
  • Night sweats
  • Palpitations
  • Panic Attacks
  • Racing heartbeat
  • Restless leg
  • Seizures / grand mal unconscious convulsions
  • Shortness of breath (dyspnea)
  • Skipped heartbeats
  • Sleep arousal
  • Slow pulse / flip-flop palpitations / skipped heartbeats
  • Sneezing
  • Sudden cardiac arrest syndromes
  • Sore legs

Thrombo Diagnosis

  • History: fluttering / flip-flop palpitations, night sweats with racing heartbeats, insomnia, panic attacks with choking, coughing, sneezing with shortness of breath, chronic fatigue, chronic bronchitis, ‘flu’ syndrome, fast weak irregular heartbeat  / slow strong 'pounding' heartbeats, peripheral neuropathy
  • Physical Examination: slow strong ventricular heartbeats mixed with fast weak atrial heartbeats, soft musical grade I – II systolic murmur, mild fever
  • Pulse Oximetry: oxygen desaturation events, elevated peripheral carboxyhemoglobin in sore extremities with phlebitis & peripheral neuropathy
  • Electrocardiogram: premature atrial beats, atrial flutter / fibrillation, premature ventricular beats, tachy-brady / brady-tachy / sick sinus syndrome
  • Echocardiogram: pulmonary valve insufficiency / tricuspid valve regurgitation
  • Arterial Blood Gas (ABG): elevated carbon dioxide saturation (hypercapnea), low oxygen saturation, elevated carboxyhemoglobin
  • Capnography: end tidal expired carbon dioxide decrease corresponds to oxygen desaturation events

Treatment / Prevention of Bloody Clots

  • Eight hours of rest / sleep every night (decreases metabolic acidosis)
  • Avoid excess drug and alcohol use
  • Adequate water intake, avoid exercise-induced dehydration
  • Diet & Nutrition: control how much and what you eat
  • Moderate aerobic exercise: golf, sex, bowling, gardening, walking, yoga, tai chi
  • Sequential venous compression treats & prevents blood clots
  • Ultrasound: helps resolve inflammation and phlebitis
  • Vibration exercise oscillates bloody clots out of heart valves

What do doctors know about thrombodextracardia, carboxyhemoglobin, or the T wave of ECG?

Thrombodextracardia is a new blood clot theory: VTE causes the sick sinus syndrome!

Blood flow generates the electric waves of the ECG, and the pulse oximeter evaluates slow venous blood flow, which leads to blood clots.

  • Cardiac muscle contractions cause blood flow.
  • Moving blood generates hemodynamic electric charges that make up the waves of the ECG.
  • Electrocardiac events move at the speed of light, which blurs the evidence.
  • Three pairs of synchronized sequential cardiac contractions generate three electric potentials that create the ECG waves called the P, QRS, and T.
  • Peristaltic contractions of the aorta and pulmonary artery create the T wave.
  • The downward outward bulging of the apex of the ventricles at the start of systole creates Q waves.
  • Old depolarization and repolarization theories need to be updated.

The importance of pulse oximetry & a new interpretation of the ECG:

  • First, the ECG can diagnose blood clots at the tricuspid or pulmonary valves.
  • Second, the pulse oximeter evaluates peripheral carboxyhemoglobin.
  • Carboxyhemoglobin locates venous acidosis and evaluates neuropathy.
  • Carboxyhemoglobin locates the origin of blood clots.
  • Blood clots at the pulmonary valve cause skipped heartbeats.
  • Skipped heartbeats lead to fainting, seizures, and sudden cardiac arrest.
  • PE of detritus causes exercise-induced asthma and narcolepsy.
  • Pulse oximeters evaluate skipped heartbeats and oxygen desaturation.
  • Cell phone monitors can predict fainting spells and emit warning signals!
  • Airline pilots, truck drivers, and train engineers can avoid accidents caused by mysterious narcolepsy or fainting spells.
  • We can all improve our life style and become happier as brain fog fades away.
  • Thank you for being curious.
 

 

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