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Blood Clot Diagnosis

  • History: fluttering / flip-flop palpitations, night sweats with racing heartbeats, insomnia, panic attacks with choking, coughing, shortness of breath (sensations of suffocation), fatigue, ‘flu’ syndrome, irregular fast / slow heartbeats
  • Physical Examination: slow strong pulse mixed with fast weak premature heartbeats (PAC’s), paradoxical soft musical grade I – II systolic heart murmurs at the pulmonary valve
  • Pulse Oximetry: oxygen desaturation events, elevated peripheral carboxyhemoglobin in sore extremities with phlebitis, peripheral neuropathy and neuropathic pain
  • Electrocardiogram: supra-ventricular tachycardia, premature atrial beats, atrial flutter / fibrillation, bradycardia, premature ventricular beats, tachy-brady / brady-tachy / sick sinus syndrome
  • Echocardiogram: pulmonary valve insufficiency / tricuspid valve regurgitation
  • Arterial Blood Gas (ABG): elevated carbon dioxide saturation, low oxygen saturation, elevated carboxyhemoglobin
  • Capnography: VTE is composed of a mixture of liquid detritus, semisolid thrombus and solid thrombus. Liquid detitus alters alveolar function and causes desaturation events (SpO2) that coincide with decreased ETCO2 changes.


Cardiac Cycle


Dr Einthoven discovered the EKG in 1903

Dr Einthoven observed three waves during each cardiac contraction. He called these waves, the P, the QRS and the T. He received the Nobel Prize in medicine/physiology in 1924.

Dr Einthoven observed that tall, narrow QRS electrical vectors corresponded to ventricular contractions. Small P waves preceding the tall waves, corresponded to atrial contractions. Wide prominent T waves followed tall spikes.

Cardiologists teach that atrial muscle cell depolarizations create small P waves, that ventricular muscle cell depolarizations create tall QRS wave; and that repolarization of ventricular cells create T waves.

A novel ECG theory by Findl and Kurtz (1077) contends that blood flow generates the ECG.

First, two atrial contractions generate blood flow electric potentials that are P waves.

Next, biventricular contractions generate blood flow electric potentials that are QRS waves.

Finally, peristaltic aortic and pulmonary artery muscle contractions generate blood flow electric potentials that are T waves.

The interpretation of the ECG needs to be reviewed and updated to make sense out of the T wave.

The EKG and Pulse

The EKG is Out of Time with the pulse oximeter during ventricular bigeminy and atrial flutter.



Vibration Therapy

Venous thromboembolism into the pulmonary valve causes right ventricular outflow tract (RVOT) obstruction. This reduces the ejection of blood from the right ventricle (RV) during systole. This decreases the ejection fraction by both ventricles and leads to RV dysplasia. The RV dilates as it weakens from dysplasia.

The dilated RV causes benign premature ventricular beats that lead to 'arrhythmogenic' cardiomyopathy and congestive heart failure (CHF). Blood clots that accumulate inside the tricuspid and pulmonary valves cause fatigue and depression.

Total body vibration platforms were developed by a Russian scientist named Vladimir Nazarov. These machines produce vibrations that separate clumps of red blood cells that occur during weightless outer space exploration.

Vertical vibrations of one G force at 20 to 50 Hertz between 0.5 to 1.5 millimeters amplitude shake loose small sticky adherent bloody clots that are stuck deep inside the cusps of the pulmonary valve.

Vibrating blood clots out of the pulmonary valve improves blood flow from the right ventricle into the pulmonary artery which stops right ventricular outflow tract (RVOT) obstruction. The RV decompresses.

Pulmonary valve function improves after blood clots are expelled out of the cusps of the valve, and the right ventricle end diastolic volume decreases which improves cardiac ejection fraction. The RV remodels and the patient becomes stronger and happier after the ECG rhythm reverts to normal sinus rhythm.

Vibration exercise can be good medicine.

Music Therapy



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